Dr. Maxwell Nartey
Professor of Symptometric Science, American School of Symptometry, NFP
World Center for Health Education & Scientific Enlightenment
The sickle cell disease is an enzymatic disorder. Plain and simple. It is not a genetic disease because a gene does not produce a disease. A gene always releases the blueprint to DNA, and this blueprint carries instructions on how to produce a specific brain essential or body essential.
A disease is pathology, and pathology is an anomaly that occurs because the three-way communication involving the genes, DNA, and the three RNAs was disrupted. This disruption occurs because forces were weakened. What weakened the forces? Poisons did; alkyl did; toxic acids and metals did, and so on and so forth.
The sickle cell disease occurs only in the individuals whose mothers ate food combinations that eliminated specific vitamins from the baby’s cells during the gestation period. Alkyl, poisons, many plant alkaloids, toxic acids, etc. eliminate vitamins.
Vitamins play a significant role in making enzymes. They are cofactors, and without them, human enzymes will not be produced.
Spectrin is the special protein that the stem cells in our red bone marrow produce to maintain the round shape of our red blood cells. Also, it is spectrin that facilitates the removal of the nucleus from the center of the red blood cells so that hemoglobin can fit into the red blood cell’s center.
Three enzymes speed up the production of spectrin. These enzymes are: FMN (flavin mononucleotide), FAD (flavin adenine dinucleotide), and PE (proteolytic enzymes). Iron and specific vitamins produce these enzymes.
Unfortunately, these three enzymes are extremely sensitive to cyanide and hydrocyanic acid. There is cyanide in corn, cassava aka manioc, plums, Lima beans, apricot, cherry, peach, pear, flax, and eucalyptus; and there is hydrocyanic acid aka prussic acid in all nuts.
Cyanide and prussic acid will denature (damage) the three above-mentioned enzymes, thereby making it impossible for the baby of a woman who eats cyanide-laden foods to produce spectrin.
Why is the sickle cell disease or the sickle cell trait common among Africans, or among people of African heritage? The answer is, no effort has been made to eliminate this inherited enzymatic disorder from the gene pool of the African lineage. The reason this inherited enzymatic disorder has not been removed is, medical science journals keep spreading the falsehood about the incurability of the sickle cell disease. It is now time to shine some floodlight on the verifiable truth about the sickle cell disease that was swept under the rug.
Medical scientists have never mentioned spectrin, FMN, FAD and PE in connection with their sickle cell research. NEVER. Therefore, leaving out the verifiable facts about a disease, and conditioning people’s minds to accept the incurability of a disease, is simply not fair to the health-conscious individuals who are seeking the total truth about a disease.
Cyanide is a poison, but it can be detoxified in a person whose liver produces the enzyme called TST (Thiosulfate Sulfur Transferase). A pregnant woman who eats cyanide foods occasionally, and produces TST, and spectrin, will be able to automatically pass on the blueprint for TST and spectrin to her baby’s genes. As a result, her baby will NEVER have the sickle cell disease or the sickle cell trait.
Cellular health boils down to the food enzymes and the ancillary enzymes we produce or do not produce. Contrary to popular belief, sickle cell has absolutely nothing to do with a person’s race. More importantly, it is curable. Also, there is no such thing as the gene for sickle cell.
The gene encodes a protein by always releasing the blueprint for that protein. Therefore, if red blood cells are crooked, it is because the person’s genes did not encode spectrin. Why didn’t the genes encode spectrin? The answer is, certain conditions were not met.
To satisfy the requirements to produce spectrin, the genes must be aligned with DNA, and DNA must also be aligned with messenger RNA so that transfer RNA and ribosomal RNA can start communicating with each other to produce spectrin, according to the specifications in the blueprint. This is what encoding a protein means.
A sickle cell carrier who eats the wrong foods, and cannot produce FMN, FAD, PE, TSF and spectrin, will never be able to cure their disease. Also, they could pass on their enzymatic disorder to one of their offspring. Contact Symptometry for what should be done to remove this awful hereditary enzymatic disorder from the lineage.
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