Understanding Crohn’s Disease

Dr. Maxwell Nartey

Professor of Symptometric Science, American School of Symptometry, NFP

World Center for Health Education & Scientific Enlightenment

The idea that Crohn’s disease is a granulomatous inflammatory disease of unknown origin is simply not true. Every disease has its cause, and Crohn’s disease is no exception. Here is what should be known about Crohn’s disease that has never been shared with the public until now.

Our colon bacteria play an important role in the production of bile. This is because they produce three vital acids. These acids are: deoxycholic acid, lithocholic acid and ursodeoxycholic acid. It is these three acids that thicken bile and make it highly concentrated. 

Since bile is extremely concentrated, the gallbladder will squeeze only one drop of it into the duodenum to facilitate the digestion of the bolus (large complex molecule made of water, acids, alkali, amylase, starch, sugar, fat, prolamins, acrylamides, oil and protein).

Therefore, if enough concentrated bile is not available for digestion, digestion will be extremely slow or indigestion will occur. Also, a person who keeps diluting the bolus by drinking water while eating, will make it extremely difficult for the food enzymes and the ancillary enzymes to thoroughly digest the bolus.

If digestion is slow, the colon bacteria will not produce their essentials on time, and there will be a hefty price to pay for this delay.

For example, instead of the bolus spending only one hour in the stomach, it will spend between three and five hours. This delay prevents fecal matter from being produced on time. 

If there is enough pepsin and HCL (hydrochloric acid) in the stomach, the digestion of the bolus is not supposed to exceed one hour. 

From the stomach, the bolus moves to the duodenum where it is supposed to spend about 30 minutes. As soon as digestion ends, the production of fecal matter must begin without delay, and this takes place two hours after eating. 

Nibblers or people who eat one hour after they have just finished eating, will cause a lot of confusion in their gastrointestinal tract.

At about two hours after eating, the colon bacteria are supposed to start producing deoxycholic acid, lithocholic acid and ursodeoxycholic acid so that the gallbladder can have them on time for the next meal. Therefore, if a person’s bad eating habits prevent their colon bacteria from shipping their acids to the gallbladder, the acids they produced will not go anywhere. They will sit in their colons.

What do colon acids do? They tear down the walls of the large intestine.

The walls of the large intestine are made of viscus (plural viscera). All the nerves for peristalsis (wavelike movement) are embedded in viscus. Also, the mechanisms for producing hypotonic and hypertonic regulators are also imbedded in viscus.

It is the hypertonic regulators that make enough water available to the colons so that fecal matter is moist and easy to evacuate after minimal straining. Then, it is the hypotonic regulators that remove excess water from the colons so that diarrhea does not occur.

Therefore, if the hypotonic regulators cannot properly send or receive signals because acids are tearing viscus down, the person will have diarrhea. This is the root cause of diarrhea associated with Crohn’s disease. What about constipation?

If the hypertonic regulators cannot function properly because acids are tearing down viscus, peristalsis will not occur. As a result, the person will be constipated. These are the underlying causes of constipation or diarrhea. If bacteria cause diarrhea, it is because they succeeded in dismantling the hypotonic regulators.

People should understand the underlying cause of constipation and diarrhea from this perspective instead of just driving to the nearest pharmacy to purchase a laxative or a stool softener.

Tearing down the walls of the large intestine will also make it impossible for cortisol to be available to the large intestine. It is the non-availability of cortisol that predisposes a person to inflammation. 

Colitis is the term for an inflamed colon, and pain will be experienced by a person whose colons are inflamed. Why? The answer is, the materials that are used to produce cortisol are the same materials that our cells use to produce substance P, enkephalin and endorphin to prevent pain.

Also, tearing down the walls of the large intestine will cause ulcers in the colons. Ulcerative colitis is the term for inflamed ulcers in the colons. The colon acids may start tearing down the duodenum, and the stomach linings to cause stomach ulcers and duodenal ulcers; ulcers in the gastrointestinal tract, and abscesses. Eventually, the wall of the large intestine will become extremely thick. 

GERD, or acid reflux associated with Crohn’s disease, should never have been a mystery. It is the colon acids that cause part of it.

In truth, Crohn’s disease is the disease that affects only the individuals who need to know more about the gallbladder, good eating habits, and how colon bacteria play a role in producing concentrated bile acids for a smooth and an efficient digestion.

It is not recommended that a health-conscious person consume alcohol. Here is why. Alcohol denatures our food enzymes, ancillary enzymes, and kinase and cyclin, the two enzymes that divide our cells. Also, many other foods and beverages should be avoided or steps should be taken to make them cell friendly.

A doctor of Symptometry should be consulted to put an end to Crohn’s disease. If this step is not taken, the colon acids may also destroy the small intestine, thereby disrupting nutrient absorption and causing all kinds of food allergies.

Managing Crohn’s disease by becoming a vegan, a vegetarian, a keto diet practitioner, etc. completely misses the point. Also, staying away from certain foods without any scientific justification, is another misstep that should be corrected. 

Finally, self-medicating with herbs, meditation, minerals and vitamins would make the disease worse. The best advice is, understand the underlying cause of Crohn’s disease and seek expert guidance to remove its source. Symptometry is available for such guidance.

© Copyright 2021, The American School of Symptometry, NFP. No part of this publication may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage and retrieval system without the written permission of The American School of Symptometry, NFP. Library of Congress copyright number Txu 1-621-370, Washington D.C.